Xbox One games will require a one-time activation code to use, but you'll still be able to trade and sell them online, Microsoft tells Kotaku?although we're not 100% clear on the details.
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Look at the prevalence of Wolfson's audio chips today, in everything from audiophile DACs to smartphones like the Exynos-powered Galaxy S III and Galaxy S 4, and it's hard not to be impressed. Factor in the company's humble beginnings in 1984 as a university offshoot in Edinburgh, Scotland, and the growth story becomes even more dramatic. The company shipped its billionth chip in 2008, its 2 billionth in 2012 and now expects to sell a billion per year by 2015.
It's ironic, then, that at the start of its journey into mobile devices Wolfson actually failed to grow quickly enough, resulting in the loss of its biggest and most high-profile customer. With Apple using its chips in a number of iPods, the Scottish company just couldn't scale up to meet a sudden rush of demand. It missed some deadlines and Cuptertino left it out of the iPod Classic as it shifted its loyalty to Cirrus Logic, where it has stayed ever since. How did it recover? Where is it headed next? And how will it break Qualcomm's continuing grip on smartphone audio in the US? Read on for answers from CEO Mike Hickey.
Filed under: Cellphones, Portable Audio/Video, Tablets, Mobile
Source: http://feeds.engadget.com/~r/weblogsinc/engadget/~3/je2QMzx0EAI/
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Northridge?s LiveRez-powered website helps it take bookings online 24/7.
Northridge Vacation Rentals
Location: Sandpoint, ID
Website: NorthridgeVacationRentals.com
Quick Quote:? ?LiveRez was amazing at helping us get started from the beginning ? not just with our website and reservation system, but with our whole business. They helped us with tips on best practices, and just went above and beyond anything I ever expected.?
The town of Sandpoint in Northern Idaho is a special place. If you don?t believe that, check out its impressive resume.
With a small population of around 8,000 people, Sandpoint is somewhat of a throwback to a simpler time.
That?s what makes it such a perfect fit for Jeremy Brown and Northridge Vacation Rentals. Like the town it manages properties in, Northridge Vacation Rentals is not huge, but growing. It?s inherit value is not in the quantity of homes it manages, but in the care it puts into each one and the level of service it offers every guest.
You see, Brown follows a very simple rule in managing homes ? call it the golden rule of property management. He manages each house like he would if it was his own.
?What we always try to think of is if we owned the property, how would we want it managed,? said Brown, who ? along with his business partner, Mike Robinson ? brings decades of experience both owning and managing properties to the company.
Jeremy Brown and Northridge Vacation Rentals will care for your home like it was their own.
Northridge Vacation Rentals? over-the-top focus on home care should come as no surprise when you learn about the company?s roots.? The business didn?t actually start renting out homes until May of 2010. Before that time it focused exclusively on the home watch and home care side of its business, and it?s still a big part of their company.
Since offering vacation rentals in 2010, the company has slowly expanded its inventory to seven Sandpoint vacation homes and condos, with more on the way. This level of inventory allows Northridge Vacation Rentals to offer exceptional service to guests and maximize each owner?s investment.
As part of this ?golden rule? philosophy, Brown makes every attempt to be transparent in his commission agreements with owners. He takes a ?no surprises? approach to end of the month owner statements. As an owner with Northridge Vacation Rentals, you won?t see a bunch of extra fees. If you agreed to a certain commission level, that?s what you get. Owners are only included in additional programs, like paid marketing plans, if they expressly give their permission. It?s the way Brown would want it as an owner.
?Unless there?s been some requested repairs or annual fee cleanings, there?s typically nothing else that comes out of their commission, unless they sign up for it,? Brown said. ?It?s pretty plain and upfront and forward.?
Northridge Vacation Rentals also takes a different stance on restricting owners from staying at their homes. While some management companies reserve certain periods of time exclusively for guest reservations, Northridge is different.
You?ll feel like you?re on top of the world hiking outside Sandpoint. The mountain ridges give you the opportunity to see three different states and Canada.
?We?re really flexible with our rental program,? Brown said. ?We never restrict how much owners can use their properties. ?
Even with allowing homeowners to use their homes whenever they want to, Brown is still able to help them maximize their investments. One particular success story is when Northridge Vacation Rentals took on a property that was previously managed by a larger competitor and increased the owner?s income by more than 30 percent in the first year.
?We?re a smaller company, so we?re able to focus on the individual property and promote and push it a lot harder than if we had a mass of 100 properties,? Brown said. ?So, there?s a lot more focus per property.?
It was an impressive feat, considering the first year managing a property is harder because you don?t have established repeat guests.
?We kept all the neighbors happy, and got a lot more renters through the property,? Brown said. ?It worked out great.?
Brown promotes a strict good neighbor policy. He knows that being able to rent out vacation homes and condos is a privilege that some other cities and towns in the United States are starting to take away. And, he knows the answer to keeping that from happening in Sandpoint is ensuring that his guests understand how to have fun while respecting their new neighbors.
This waterfront home off nearby Priest Lake allows guests to enjoy the natural beauty of Northern Idaho.
?From the beginning, we?ve always taken on the harder-to-manage properties,? Brown said. ?You just have to definitely keep on top of the problems and maintain peace among the neighbors that live within the block. If you have rowdy renters, you?ll hear about it.?
This philosophy has helped him gain favor (and business) from some of the condo and home owner associations in Sandpoint. And, because he?s earned their trust, Brown takes his job very seriously.
?We currently manage two home owners associations that we also manage rentals within,? Brown said. ?So, we obviously have to keep the owner of the vacation rental property happy, and we also have to make sure that all the other home owners are happy. We definitely have a lot to lose if we do a poor job.?
In addition to its small-town charm, Sandpoint and the surrounding area are home to some of the most beautiful landscapes in all the United States. It?s an outdoor wonderland, whether you?re enjoying the waters of gorgeous Lake Pend Oreille that sits adjacent to the town, hiking through the rugged terrain of Scotchman Peaks Wilderness, or experiencing the fresh powder at Idaho?s largest ski resort at Schweitzer Mountain.
Nearby LakePend Oreille is 43 miles long and the fifth deepest lake in the United States at 1,150 feet deep. With the lake having been carved by glaciers during the ice age, it?s no surprise that majestic mountains seem to rise directly out of its deep blue waters.
Sportsmen will be pleased to know that the lake is home to two of Idaho?s longest standing state records for the trout family. Nelson Higgins? 32-pound record Dolly Varden has stood since 1949, and Wes Hamlet?s 37-pound Kamloops Rainbow Trout has stood since 1947.? According to Brown, anglers are still pulling monster fish from the water of Lake Pend Oreille. In fact, he said the winning Rainbow in the most recent K&K Spring Fishing Derby weighed in at more than 18 pounds and the winning Mackinaw was more than 26 pounds.
You?re bound to run into native wildlife and breathtaking views hiking through Scotchman Peaks Wilderness.
The lake is a beautiful site from atop the ski runs at Schweitzer Mountain Resort. But, it?s not the only thing you?ll see. At 6,400 feet elevation, you can view three states, Canada, and three mountain ranges. In the winter months you can enjoy spacious runs, with more than 2900 skiable acres ? even more than its world-renown neighbor to the southeast, Sun Valley.
Sandpoint also offers a thriving arts community and boasts a wide ranges of festivals throughout the year. No matter the time of year, there?s truly always something fun to do in Sandpoint.
?I think that we?re just really, really blessed that we get to live in such an amazing area,? said Brown, a third-generation lifelong resident of Sandpoint.
And if you?re planning a trip to Sandpoint, you should check out Northridge Vacation Rentals? beautiful website, where you can book online ? just like a hotel ? 24 hours a day, 7 days a week. It?s filled with a wealth of information on the properties available through the company?s rental program, along with information on the area and on services the company offers owners.
Another thing that is available 24/7 is Northridge?s vacation rental software. That?s an important detail for the company, because they pride themselves on responding to issues around the clock.
When Brown and the team at Northridge Vacation Rentals first started managing vacation rentals, they stayed local, choosing Boise-based LiveRez.
?LiveRez was amazing at helping us get started from the beginning ? not just with our website and reservation system, but with our whole business,? Brown said. ?They helped us with tips on best practices, and just went above and beyond anything I ever expected.?
And now, nearing their three-year anniversary with the company they continue to jump on the new technology offered by LiveRez. They?re so plugged into their software that they even offer to beta test new features before they come out.
One new piece of technology that?s really made a big difference is the LiveRez CRM system, which can automate the bulk of a company?s communications with guests and owners. And while Brown doesn?t have hundreds of properties to handle, he also doesn?t have a huge staff. The business is staffed by him, his business partner, Mike, and their wives. And, with the level of detail that they give each home and each guest, they don?t have time to sit in the office and do repetitive tasks.
You can stay right on the water with Northridge Vacation Rentals.
?It?s really freed up a lot of time for me,? Brown said of the CRM system. ?I really enjoy it. It?s helped a lot.?
?I think smaller companies would need it more than other companies,? he continued. ?I don?t have a huge team of reservationists and secretaries and people to handle these things. It?s all just a few people. And the more time I sit behind the computer is less time I spend out in the field looking over properties and talking to the home owners.?
LiveRez has not only saved the company time, but also helped them grow their business and make more money.
?We increased what we earn on each property every year since the beginning,? Brown said. ?So, every year each property makes more and more, so it?s definitely been a steady, upward growth.?
LiveRez?s world-class software helps Northridge Vacation Rentals offer world-class service, and stay competitive, even in the face of larger, more established competitors ? a challenge not much different than the one LiveRez faced when it first started.
So, maybe it comes as no surprise that the partnership between these two Idahoans has resulted in the growth of both businesses. But, isn?t that what a partnership is really about?
?I would recommend LiveRez to anyone,? Brown said, ??besides my competitors.?
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Public release date: 20-May-2013
Share ] Contact: Nathaniel Dunford
ndunford@thoracic.org
American Thoracic Society
ATS 2013, PHILADELPHIA ? Both fine-particle air pollution and noise pollution may increase a person's risk of developing cardiovascular disease, according to German researchers who have conducted a large population study, in which both factors were considered simultaneously.
"Many studies have looked at air pollution, while others have looked at noise pollution," said study leader Barbara Hoffmann, MD, MPH, a professor of environmental epidemiology at the IUF Leibniz Research Institute for Environmental Medicine in Germany. "This study looked at both at the same time and found that each form of pollution was independently associated with subclinical atherosclerosis."
The research will be presented at ATS 2013.
"This study is important because it says that both air pollution and noise pollution represent important health problems," said Dr. Philip Harber, a professor of public health at the University of Arizona who was not involved in the research. "In the past, some air pollution studies have been dismissed because critics said it was probably the noise pollution that caused the harm, and vice versa. Now we know that people who live near highways, for instance, are being harmed by air pollution and by noise pollution."
Using data from the Heinz Nixdorf Recall study, an ongoing population study from three neighboring cities in the Ruhr region of Germany, Dr. Hoffmann and her colleagues assessed the long-term exposure to fine particulate matter with an aerodynamic diameter
The exposure to air pollutants was calculated using the EURopean Air Pollution Disperson, or EURAD, model. Exposure to traffic noise was calculated using European Union models of outdoor traffic noise levels. These levels were quantified as weighted 24-hour mean exposure (Lden) and nighttime exposure (Lnight).
To determine the association of the two variables with cardiovascular risk, the researchers looked at thoracic aortic calcification (TAC), a measure of subclinical atherosclerosis.
TAC was quantified using non-contrast enhanced electron beam computed tomography. Using multiple linear regression, the researchers controlled for other cardiovascular risk factors, including age, gender, education, unemployment, smoking status and history, exposure to second-hand smoke, physical activity, alcohol use and body mass index.
After controlling for these variables, the researchers found that fine-particle air pollution was associated with an increase in TAC burden by 19.9 % (95%CI 8.2; 32.8%) per 2.4g/m3. (To put that increase in perspective: in the United States, the Environmental Protection Agency recently revised the overall limit downward from 15 to 12g/m 3).
The researchers also found that nighttime traffic noise pollution increased TAC burden by 8% (95% CI 0.8; 8.9%) per 5 dB. (An average living room would typically have a noise level of about 40 A-weighted decibels, or dB(A), an expression of the relative loudness of sounds as perceived by the human ear, while busy road traffic would generate about 70-80dB(A)). Mean exposure to traffic noise over 24 hours was not associated with increased TAC.
Among subgroups of participants, the researchers found even stronger associations. The interaction of PM2.5 and TAC was clearer among those younger than 65, participants with prevalent coronary artery disease and those taking statins. In contrast, the effect of Lnight was stronger in participants who were not obese, did not have coronary artery disease and did not take statins.
Although the cross-sectional design of this study limits the causal interpretation of the data, Dr. Hoffmann said, "both exposures seem to be important and both must be considered on a population level, rather than focusing on just one hazard."
She added that her research group plans to conduct a longitudinal analysis with repeated measures of TAC over time.
###
* Please note that numbers in this release may differ slightly from those in the abstract. Many of these investigations are ongoing; the release represents the most up-to-date data available at press time.
Abstract 40036
Searching For The Culprit: Is Urban Particulate Air Pollution Or Road Traffic Noise Responsible For The Association Of Traffic Proximity With Subclinical Atherosclerosis - Results From The Heinz Nixdorf Recall Study
Type: Scientific Abstract
Category: 06.01 - Air Pollution: Epidemiology and Mechanisms (EOH)
Authors: B. Hoffmann1, F. Hennig2, S. Moebus3, S. Mohlenkamp4, N. Dragano5, H. Jakobs6, R. Erbel4, K.-H. Jockel3, H. Kalsch4; 1IUF Leibniz Research Institute for Environmental Medicine - Dusseldorf/DE, 2IUF Leibniz Research Institute for Environmental Medicine - Dsseldorf/DE, 3University of Duisburg-Essen - Essen/DE, 4West-German Heart Center - Essen/DE, 5University of Duesseldorf - Duesseldorf/DE, 6Rhenish Institute for Environmental Research - Cologne/DE
Abstract Body
Rationale: Long-term particulate matter (PM) air pollution and high ambient noise levels are traffic-related environmental exposures, which often occur concurrently. Traffic proximity has been linked to subclinical atherosclerosis; however until now, no study has investigated, whether PM or road traffic noise is responsible for this association. We aimed to investigate the independent associations of PM and road traffic noise with thoracic aortic calcification (TAC), a measure of subclinical atherosclerosis.
Methods: We used baseline data (2000-2003) from the German Heinz Nixdorf Recall Study, a population-based cohort of 4814 participants. We assessed long-term exposure to fine particulate matter with an aerodynamic diameter Results: In 4238 included participants (mean age 60 years, 49.9% male), TAC score had a highly skewed distribution with a median Agatston-score of 20.04 (IQR: 143.31). PM2.5, and Lnight were independently associated with increasing TAC-burden (19.9% [95%CI 8.2; 32.8%] per 2.4g/m3 PM2.5, and 4.8% [95%CI 0.8; 8.9%] per 5 dB Lnight, respectively), while Lden was not associated with TAC. The effect of PM on TAC was not modified by high traffic noise exposure and vice versa. We observed an interaction of PM2.5 with several personal participant characteristics, displaying clearly stronger associations for younger participants (Conclusions: Long-term exposure to fine PM air pollution and night-time road traffic noise are independently associated with thoracic aortic calcification, a measure of subclinical atherosclerosis.
[ | E-mail | Share ] ?
AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.
Public release date: 20-May-2013 Share ] Contact: Nathaniel Dunford
ndunford@thoracic.org
American Thoracic Society
ATS 2013, PHILADELPHIA ? Both fine-particle air pollution and noise pollution may increase a person's risk of developing cardiovascular disease, according to German researchers who have conducted a large population study, in which both factors were considered simultaneously.
"Many studies have looked at air pollution, while others have looked at noise pollution," said study leader Barbara Hoffmann, MD, MPH, a professor of environmental epidemiology at the IUF Leibniz Research Institute for Environmental Medicine in Germany. "This study looked at both at the same time and found that each form of pollution was independently associated with subclinical atherosclerosis."
The research will be presented at ATS 2013.
"This study is important because it says that both air pollution and noise pollution represent important health problems," said Dr. Philip Harber, a professor of public health at the University of Arizona who was not involved in the research. "In the past, some air pollution studies have been dismissed because critics said it was probably the noise pollution that caused the harm, and vice versa. Now we know that people who live near highways, for instance, are being harmed by air pollution and by noise pollution."
Using data from the Heinz Nixdorf Recall study, an ongoing population study from three neighboring cities in the Ruhr region of Germany, Dr. Hoffmann and her colleagues assessed the long-term exposure to fine particulate matter with an aerodynamic diameter
The exposure to air pollutants was calculated using the EURopean Air Pollution Disperson, or EURAD, model. Exposure to traffic noise was calculated using European Union models of outdoor traffic noise levels. These levels were quantified as weighted 24-hour mean exposure (Lden) and nighttime exposure (Lnight).
To determine the association of the two variables with cardiovascular risk, the researchers looked at thoracic aortic calcification (TAC), a measure of subclinical atherosclerosis.
TAC was quantified using non-contrast enhanced electron beam computed tomography. Using multiple linear regression, the researchers controlled for other cardiovascular risk factors, including age, gender, education, unemployment, smoking status and history, exposure to second-hand smoke, physical activity, alcohol use and body mass index.
After controlling for these variables, the researchers found that fine-particle air pollution was associated with an increase in TAC burden by 19.9 % (95%CI 8.2; 32.8%) per 2.4g/m3. (To put that increase in perspective: in the United States, the Environmental Protection Agency recently revised the overall limit downward from 15 to 12g/m 3).
The researchers also found that nighttime traffic noise pollution increased TAC burden by 8% (95% CI 0.8; 8.9%) per 5 dB. (An average living room would typically have a noise level of about 40 A-weighted decibels, or dB(A), an expression of the relative loudness of sounds as perceived by the human ear, while busy road traffic would generate about 70-80dB(A)). Mean exposure to traffic noise over 24 hours was not associated with increased TAC.
Among subgroups of participants, the researchers found even stronger associations. The interaction of PM2.5 and TAC was clearer among those younger than 65, participants with prevalent coronary artery disease and those taking statins. In contrast, the effect of Lnight was stronger in participants who were not obese, did not have coronary artery disease and did not take statins.
Although the cross-sectional design of this study limits the causal interpretation of the data, Dr. Hoffmann said, "both exposures seem to be important and both must be considered on a population level, rather than focusing on just one hazard."
She added that her research group plans to conduct a longitudinal analysis with repeated measures of TAC over time.
###
* Please note that numbers in this release may differ slightly from those in the abstract. Many of these investigations are ongoing; the release represents the most up-to-date data available at press time.
Abstract 40036
Searching For The Culprit: Is Urban Particulate Air Pollution Or Road Traffic Noise Responsible For The Association Of Traffic Proximity With Subclinical Atherosclerosis - Results From The Heinz Nixdorf Recall Study
Type: Scientific Abstract
Category: 06.01 - Air Pollution: Epidemiology and Mechanisms (EOH)
Authors: B. Hoffmann1, F. Hennig2, S. Moebus3, S. Mohlenkamp4, N. Dragano5, H. Jakobs6, R. Erbel4, K.-H. Jockel3, H. Kalsch4; 1IUF Leibniz Research Institute for Environmental Medicine - Dusseldorf/DE, 2IUF Leibniz Research Institute for Environmental Medicine - Dsseldorf/DE, 3University of Duisburg-Essen - Essen/DE, 4West-German Heart Center - Essen/DE, 5University of Duesseldorf - Duesseldorf/DE, 6Rhenish Institute for Environmental Research - Cologne/DE
Abstract Body
Rationale: Long-term particulate matter (PM) air pollution and high ambient noise levels are traffic-related environmental exposures, which often occur concurrently. Traffic proximity has been linked to subclinical atherosclerosis; however until now, no study has investigated, whether PM or road traffic noise is responsible for this association. We aimed to investigate the independent associations of PM and road traffic noise with thoracic aortic calcification (TAC), a measure of subclinical atherosclerosis.
Methods: We used baseline data (2000-2003) from the German Heinz Nixdorf Recall Study, a population-based cohort of 4814 participants. We assessed long-term exposure to fine particulate matter with an aerodynamic diameter Results: In 4238 included participants (mean age 60 years, 49.9% male), TAC score had a highly skewed distribution with a median Agatston-score of 20.04 (IQR: 143.31). PM2.5, and Lnight were independently associated with increasing TAC-burden (19.9% [95%CI 8.2; 32.8%] per 2.4g/m3 PM2.5, and 4.8% [95%CI 0.8; 8.9%] per 5 dB Lnight, respectively), while Lden was not associated with TAC. The effect of PM on TAC was not modified by high traffic noise exposure and vice versa. We observed an interaction of PM2.5 with several personal participant characteristics, displaying clearly stronger associations for younger participants (Conclusions: Long-term exposure to fine PM air pollution and night-time road traffic noise are independently associated with thoracic aortic calcification, a measure of subclinical atherosclerosis.
[ | E-mail | Share ] ?
AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.
Source: http://www.eurekalert.org/pub_releases/2013-05/ats-sfa051313.php
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?By The Associated PressThis aerial photo shows the remains of houses in Moore, Okla., following a tornado Monday, May 20, 2013. A tornado roared through the Oklahoma City suburbs Monday, flattening entire neighborhoods, setting buildings on fire and landing a direct blow on an elementary school. (AP Photo/Steve Gooch)
This aerial photo shows the remains of houses in Moore, Okla., following a tornado Monday, May 20, 2013. A tornado roared through the Oklahoma City suburbs Monday, flattening entire neighborhoods, setting buildings on fire and landing a direct blow on an elementary school. (AP Photo/Steve Gooch)
Glenn Rusk hugs his neighbor Sherie Loman outside her home north of Briarwood Elementary School after a tornado moved through the area, in Moore, Okla., Monday, May 20, 2013. (AP Photo/The Oklahoman, David McDaniel)
A woman carries a child through a field near the collapsed Plaza Towers Elementary School in Moore, Okla., Monday, May 20, 2013. The relationship between the woman and the child was not immediately known. A tornado as much as a mile (1.6 kilometers) wide with winds up to 200 mph (320 kph) roared through the Oklahoma City suburbs Monday, flattening entire neighborhoods, setting buildings on fire and landing a direct blow on an elementary school. (AP Photo Sue Ogrocki)
JoAnn Anderson sorts through the rubble of her home after a tornado on Monday, May 20, 2013 in Moore, Okla. A monstrous tornado roared through the Oklahoma City suburbs, flattening entire neighborhoods with winds up to 200 mph, setting buildings on fire and landing a direct blow on an elementary school. (AP Photo/The Oklahoman, Steve Sisney)
Teachers carry children away from Briarwood Elementary school after a tornado destroyed the school in south Oklahoma City, Monday, May 20, 2013. A monstrous tornado roared through the Oklahoma City suburbs, flattening entire neighborhoods with winds up to 200 mph, setting buildings on fire and landing a direct blow on an elementary school. (AP Photo/The Oklahoman, Paul Hellstern)
The devastating tornado that swept through Oklahoma turned houses into matchsticks and sent parents and teachers running to pull children from the wreckage of an elementary school in the eye of the storm. At least 51 people were killed by the 200-mph storm and dozens of homes destroyed. The toll was expected to rise. Here are images from the aftermath :
Associated Pressatherosclerosis steven tyler tropic thunder carnie wilson missing reese witherspoon pregnant billy joel
12 hours ago
REUTERS
This custom-made electric guitar played by the late John Lennon and George Harrison of the Beatles recently sold at auction.
A custom-made electric guitar played by the late John Lennon and George Harrison of the Beatles sold at a New York auction on Saturday for $408,000, said officials with the company behind the event.
The semi-hollow-body guitar, manufactured by the VOX company, was sold to an unidentified U.S. buyer at the "Music Icons" event organized by Beverly Hills, California-based Julien's Auctions and held at the Hard Rock Cafe in Manhattan.
Julien's said previously it expected the guitar, which was the centerpiece of Saturday's sale, to fetch between $200,000 and $300,000.
Harrison played the instrument, distinguished by two symmetrical flared shoulders on the upper body, while practicing "I Am The Walrus," and Lennon used it in a video session for the song "Hello, Goodbye," according to a statement from Julien's Auctions.
Both songs were on the Beatles' 1967 album "Magical Mystery Tour."
The VOX guitar was a prototype instrument custom-built for Lennon in 1966, said Martin Nolan, executive director of Julien's. Lennon gave the VOX guitar as a gift in 1967 to Yanni "Magic Alex" Mardas, who was the electronics engineer for the band's Apple Records label, the auction house said.
The instrument, displayed in recent weeks at a museum in Ireland before the sale, was sold a few years ago by Christie's Auction House for a little over $100,000. Nolan said the latest buyer, who sent a representative to Saturday's auction to bid on his behalf, wished to remain anonymous.
Lennon was shot to death in New York in 1980 by a deranged fan, and Harrison died of lung cancer in Los Angeles in 2001. The surviving members of the Beatles are Paul McCartney and Ringo Starr.
Copyright 2013 Thomson Reuters.
Source: http://www.today.com/entertainment/beatles-guitar-auctioned-tune-408-000-1C9984548
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Public release date: 19-May-2013 Share ] Contact: Catherine Kolf
ckolf@jhmi.edu
443-440-1929
Johns Hopkins Medicine
Scientists at Johns Hopkins have turned their view of osteoarthritis (OA) inside out. Literally. Instead of seeing the painful degenerative disease as a problem primarily of the cartilage that cushions joints, they now have evidence that the bone underneath the cartilage is also a key player and exacerbates the damage. In a proof-of-concept experiment, they found that blocking the action of a critical bone regulation protein in mice halts progression of the disease.
The prevailing theory on the development of OA focuses on joint cartilage, suggesting that unstable mechanical pressure on the joints leads to more and more harm to the cartilageand pain to the patientuntil the only treatment option left is total knee or hip replacement. The new theory, reported May 19 in Nature Medicine, suggests that initial harm to the cartilage causes the bone underneath it to behave improperly by building surplus bone. The extra bone stretches the cartilage above and speeds its decline.
"If there is something wrong with the leg of your chair and you try to fix it by replacing the cushion, you haven't solved the problem," says Xu Cao, Ph.D., director of the Center for Musculoskeletal Research in the Department of Orthopaedic Surgery at the Johns Hopkins University School of Medicine. "We think that the problem in OA is not just the cartilage 'cushion,' but the bone underneath," he adds.
Joints are formed at the intersection of two bones. To prevent the grinding and wearing down of the ends of the bones, they are capped with a thin layer of cartilage, which not only provides a smooth surface for joint rotation but also absorbs some of the weight and mechanical strain placed on the joint. The degeneration of this protective layer causes extreme pain leading to limited mobility.
Cao says degeneration is most frequently initiated by instability in the load-bearing joints of the knee and hip caused by injury or strain, so athletes, overweight people and people whose muscles are weakened by aging are at highest risk of developing OA. The prevalence of the disease is rapidly increasing; it currently affects 27 million Americans and may double by 2030. The only treatment available is pain management, or surgical replacement of the arthritic joint with a prosthetic one.
Cao says that the lack of effective drugs or a complete understanding of the underlying process that causes OA to progress led his group to search for a different underlying cause. "We began to think of cartilage and the bone underneath it, called subchondral bone, as functioning as a single unit," says Cao. "That helped us to see the ways in which the bone was responding to changes in the cartilage and exacerbating the problem."
Using mice with ACL (anterior cruciate ligament) tears, which are known to lead to OA of the knee, the researchers found that, as soon as one week after the injury, pockets of subchondral bone had been "chewed" away by cells called osteoclasts. This process activated high levels in the bone of a protein called TGF-beta1, which, in turn, recruited stem cells to the site so that they could create new bone to fill the holes. Cao calls these pockets of new bone formation "osteoid islets."
But the bone building and the bone destruction processes were not coordinated in the mice, and the bone building prevailed, placing further strain on the cartilage cap. It is this extraneous bone formation that Cao and his colleagues believe to be at the heart of OA, as confirmed in a computer simulation of the human knee.
With this new hypothesis in hand, complete with a protein suspect, the team tried several methods to block the activity of TGF-beta1. When a TGF-beta1 inhibitor drug was given intravenously, the subchondral bone improved significantly, but the cartilage cap deteriorated further. However, when a different inhibitor of TGF-beta1, an antibody against it, was injected directly into the subchondral bone, the positive effects were seen in the bone without the negative effects on the cartilage. The same result was also seen when TGF-beta1 was genetically disrupted in the bone precursor cells alone.
"Our results are potentially really good news for patients with OA," says Cao. "We are already working to develop a clinical trial to test the efficacy of locally applied TGF-beta1 antibodies in human patients at early stages of OA." If successful, their nonsurgical treatment could make OA and the pain and debilitation it causes halt in its tracks, he says.
###
Other authors of the report include Gehua Zhen, Xiaofeng Jia, Janet Crane, Simon Mears, Frederic Askin, Frank Frassica, Weizhong Chang, John Carrino, Andrew Cosgarea, Dmitri Artemov, Lee Riley, Paul Sponseller and Mei Wan of the Johns Hopkins University School of Medicine; Chunyi Wen, Jie Yao and William Weijia Lu of the University of Hong Kong; and Yu Li, Qianming Chen, Zhihe Zhao and Xuedong Zhou of Sichuan University.
This work was supported by grants from the National Institute of Diabetes and Digestive and Kidney Diseases (DK057501, DK08098).
On the Web:
Link to article (live after embargo lifts): http://dx.doi.org/10.1038/nm.3143
Cao Lab: http://www.hopkinsortho.org/xu_cao_phd.html
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Media Contacts: Catherine Kolf; 443-287-2251; ckolf@jhmi.edu
Vanessa McMains; 410-502-9410; vmcmain1@jhmi.edu
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Public release date: 19-May-2013 Share ] Contact: Catherine Kolf
ckolf@jhmi.edu
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Johns Hopkins Medicine
Scientists at Johns Hopkins have turned their view of osteoarthritis (OA) inside out. Literally. Instead of seeing the painful degenerative disease as a problem primarily of the cartilage that cushions joints, they now have evidence that the bone underneath the cartilage is also a key player and exacerbates the damage. In a proof-of-concept experiment, they found that blocking the action of a critical bone regulation protein in mice halts progression of the disease.
The prevailing theory on the development of OA focuses on joint cartilage, suggesting that unstable mechanical pressure on the joints leads to more and more harm to the cartilageand pain to the patientuntil the only treatment option left is total knee or hip replacement. The new theory, reported May 19 in Nature Medicine, suggests that initial harm to the cartilage causes the bone underneath it to behave improperly by building surplus bone. The extra bone stretches the cartilage above and speeds its decline.
"If there is something wrong with the leg of your chair and you try to fix it by replacing the cushion, you haven't solved the problem," says Xu Cao, Ph.D., director of the Center for Musculoskeletal Research in the Department of Orthopaedic Surgery at the Johns Hopkins University School of Medicine. "We think that the problem in OA is not just the cartilage 'cushion,' but the bone underneath," he adds.
Joints are formed at the intersection of two bones. To prevent the grinding and wearing down of the ends of the bones, they are capped with a thin layer of cartilage, which not only provides a smooth surface for joint rotation but also absorbs some of the weight and mechanical strain placed on the joint. The degeneration of this protective layer causes extreme pain leading to limited mobility.
Cao says degeneration is most frequently initiated by instability in the load-bearing joints of the knee and hip caused by injury or strain, so athletes, overweight people and people whose muscles are weakened by aging are at highest risk of developing OA. The prevalence of the disease is rapidly increasing; it currently affects 27 million Americans and may double by 2030. The only treatment available is pain management, or surgical replacement of the arthritic joint with a prosthetic one.
Cao says that the lack of effective drugs or a complete understanding of the underlying process that causes OA to progress led his group to search for a different underlying cause. "We began to think of cartilage and the bone underneath it, called subchondral bone, as functioning as a single unit," says Cao. "That helped us to see the ways in which the bone was responding to changes in the cartilage and exacerbating the problem."
Using mice with ACL (anterior cruciate ligament) tears, which are known to lead to OA of the knee, the researchers found that, as soon as one week after the injury, pockets of subchondral bone had been "chewed" away by cells called osteoclasts. This process activated high levels in the bone of a protein called TGF-beta1, which, in turn, recruited stem cells to the site so that they could create new bone to fill the holes. Cao calls these pockets of new bone formation "osteoid islets."
But the bone building and the bone destruction processes were not coordinated in the mice, and the bone building prevailed, placing further strain on the cartilage cap. It is this extraneous bone formation that Cao and his colleagues believe to be at the heart of OA, as confirmed in a computer simulation of the human knee.
With this new hypothesis in hand, complete with a protein suspect, the team tried several methods to block the activity of TGF-beta1. When a TGF-beta1 inhibitor drug was given intravenously, the subchondral bone improved significantly, but the cartilage cap deteriorated further. However, when a different inhibitor of TGF-beta1, an antibody against it, was injected directly into the subchondral bone, the positive effects were seen in the bone without the negative effects on the cartilage. The same result was also seen when TGF-beta1 was genetically disrupted in the bone precursor cells alone.
"Our results are potentially really good news for patients with OA," says Cao. "We are already working to develop a clinical trial to test the efficacy of locally applied TGF-beta1 antibodies in human patients at early stages of OA." If successful, their nonsurgical treatment could make OA and the pain and debilitation it causes halt in its tracks, he says.
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Other authors of the report include Gehua Zhen, Xiaofeng Jia, Janet Crane, Simon Mears, Frederic Askin, Frank Frassica, Weizhong Chang, John Carrino, Andrew Cosgarea, Dmitri Artemov, Lee Riley, Paul Sponseller and Mei Wan of the Johns Hopkins University School of Medicine; Chunyi Wen, Jie Yao and William Weijia Lu of the University of Hong Kong; and Yu Li, Qianming Chen, Zhihe Zhao and Xuedong Zhou of Sichuan University.
This work was supported by grants from the National Institute of Diabetes and Digestive and Kidney Diseases (DK057501, DK08098).
On the Web:
Link to article (live after embargo lifts): http://dx.doi.org/10.1038/nm.3143
Cao Lab: http://www.hopkinsortho.org/xu_cao_phd.html
Media Relations and Public Affairs
Media Contacts: Catherine Kolf; 443-287-2251; ckolf@jhmi.edu
Vanessa McMains; 410-502-9410; vmcmain1@jhmi.edu
Shawna Williams; 410-955-8236; shawna@jhmi.edu
[ | E-mail | Share ] ?
AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.
Source: http://www.eurekalert.org/pub_releases/2013-05/jhm-nto051713.php
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MAIDUGURI, Nigeria (AP) ? Nigeria's military declared a 24-hour curfew Saturday on neighborhoods in a northeastern city that's the spiritual home of an Islamic extremist network as soldiers continued the government's emergency campaign in the region, with authorities saying they killed 10 suspected insurgents.
A statement Saturday on behalf of Lt. Col. Sagir Musa named 11 areas of Maiduguri where people must remain inside their homes until further notice. Musa said it was part of the military's push since President Goodluck Jonathan issued an emergency decree Tuesday allowing soldiers to arrest people at will and take over buildings suspected to house extremists in Adamawa, Borno and Yobe states.
Soldiers arrested some 65 suspected extremists who were "attempting to infiltrate Maiduguri" after military strikes on camps in a nearby forest reserve, military spokesman Brig. Gen. Chris Olukolade said in a statement Saturday. Olukolade said soldiers killed another 10 suspected extremists in Maiduguri's Gamboru neighborhood, one of the areas now under curfew.
There was no independent confirmation of the arrests nor the killings. An Associated Press journalist in Maiduguri saw roadblocks manned by soldiers in the city, as well as trucks lined up outside the city, apparently blocked by the military from entering.
Maiduguri, the capital of Borno state, once was home to the main mosque of Boko Haram. Nigeria's Islamic extremist insurgency grew out of a 2009 riot led by Boko Haram members in Maiduguri that ended in a military and police crackdown that killed some 700 people. The group's leader died in police custody in an apparent summary execution, fueling dissent that broke into the open in 2010 with the targeted killings of government officials, security agents and religious leaders who spoke out against the sect. The killings gradually morphed into the large-scale extremist network that is plaguing Nigeria today.
Soldiers backed by jet fighters and military helicopters have flooded into the northeast since Jonathan's order Tuesday. On Thursday, soldiers attacked suspected camps sheltering insurgents in a forest reserve south of Maiduguri. At least 21 suspected extremists died in the attack, an official told the AP on Friday. The military said it destroyed the extremists' equipment and gasoline supplies.
On Saturday, Olukolade asked the public to inform authorities if they saw anyone attempting to gather large quantities of gasoline.
"Some of the fleeing insurgents from various camps have been noted to be in search of fuel," the brigadier general said.
This new military campaign comes on top of a previous massive deployment of soldiers and police to the region. That deployment failed to stop violence by Islamic extremists, who have killed more than 1,600 people since 2010, according to an AP count. It also has seen soldiers arrest, torture and even kill civilians.
Boko Haram, whose name means "Western education is sacrilege" in the Hausa language of Nigeria's predominantly Muslim north, has said it wants to establish an Islamic state in Nigeria and wants the government to release all of its imprisoned followers. Boko Haram has sparked splinter groups like Ansaru, which has kidnapped foreign hostages. Analysts and diplomats also say the network has loose ties to two other al-Qaida-influenced groups on the African continent.
___
Associated Press writer Bashir Adigun in Abuja, Nigeria, contributed to this report.
___
Jon Gambrell reported from Yenagoa, Nigeria, and can be reached at www.twitter.com/jongambrellAP .
Source: http://news.yahoo.com/nigeria-military-declares-24-hour-curfew-city-151432766.html
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For those involved in auto accidents, a visit to a Pleasanton chiropractor is advised. This is sure to bring respite from any pain you may be suffering. The most common injuries suffered in a collision involve the neck and back.
Cervical acceleration-deceleration injuries are commonly experienced after a collision. These types of injury are more commonly known as whiplash and are very likely to occur when a car is hit from behind. On impact the head jerks back then snaps forward. This causes severe trauma to the vertebrae and soft tissues of the neck.
Damage to the neck is not always immediately apparent. It pays to visit a chiropractor after an auto accident even if there is little evidence of injury. It may take days or weeks for the effects to be fully felt. By visiting a chiropractor, you will be giving your body the optimum chance of rehabilitating itself.
The neck and middle spine are most commonly affected. The top two of the seven bones of the cervical vertebrae are the axis and atlas. These allow head movements back and forth as well as from side to side. Trauma to the area causes disruption to the vertebrae with associated soft tissue damage. When symptoms do start to appear, they may include pain in the back and neck. Headaches are common as are pins and needles in the legs and arms. Pain and aching may also spread to the shoulders.
The first task of the chiropractor will be to adjust the spine and allow free passage of nerves and blood vessels. Anything which compromises or squeezes the spinal column causes problems in other areas. Once the spine is in alignment, nerves and blood vessels can travel to all areas of the body as they should.
Chiropractors may use a number of therapies to help people recover after auto accidents. They may use gentle massage or acupuncture to accelerate recuperation. Hot or cold packs may be prescribed. Appropriate exercises will be recommended so that future movement will not limited. A Pleasanton chiropractor will also be able to prescribe medication to relieve pain and reduce inflammation.
Chiropractic care alleviates thigh, groin and spine pain quickly. You will get more information about an experienced Pleasanton chiropractor at http://www.siliconvalleyspinecare.com right now.
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BRIDGEPORT, Conn. (AP) ? The commuter train derailment and collision that left dozens injured outside New York City was not the result of foul play, officials said Saturday, but a fractured section of rail is being studied to determine if it is connected to the accident.
National Transportation Safety Board member Earl Weener said Saturday the broken rail is of substantial interest to investigators and a portion of the track will be sent to a lab for analysis.
Weener said it's not clear if the accident caused the fracture or if the rail was broken before the crash. He said he won't speculate on the cause of the derailment and emphasized the investigation was in its early stages.
Seventy-two people were sent to the hospital Friday evening after a Metro-North train heading east from New York City derailed and was hit by a train heading west from New Haven. Most have been discharged.
Officials earlier described devastating damage and said it was fortunate no one was killed.
"All of the injured people described the really harrowing experience of having the train jolt to a stop, the dust, darkness, other kinds of factors that made it particularly frightening," said U.S. Sen. Richard Blumenthal, who visited several patients in the hospital.
Blumenthal said a Metro-North conductor helped passengers despite her own injuries.
"Her story is really one of great strength and courage helping other passengers off the train in spite of her own very severe pain," Blumenthal said. "She eventually had to be helped off herself."
The crash damaged the tracks and threatened to snarl travel in the Northeast Corridor. The crash also caused Amtrak to suspend service between New York and Boston.
"The damage is absolutely staggering," Blumenthal said, describing the shattered interior of cars and tons of metal tossed around. "I feel that we are fortunate that even more injuries were not the result of this very tragic and unfortunate accident."
U.S. Sen. Chris Murphy said it was "frankly amazing" people weren't killed on scene.
Both said new Metro-North Railroad cars built with higher standards may have saved lives.
Metro-North said train service will remain suspended between South Norwalk and New Haven until further notice. Railroad officials said rebuilding the two tracks and restore train service "will take well into next week."
"We want our customers to know that while you travel on Metro-North, you can remain confident that your safety, and the safety of our employees, is always the first priority in everything we do," said Howard Permut, President of MTA Metro-North Railroad.
NTSB investigators arrived Saturday and are expected to be on site for seven to 10 days. They will look at the brakes and performance of the trains, the condition of the tracks, crew performance and train signal information, among other things.
When the NTSB has concluded the on-site phase of its investigation, Metro-North will begin to remove the damaged rail cars and remaining debris. The process requires specialized, heavy equipment that will be in place by Sunday, officials said. Only after the damaged train cars have been removed can Metro-North begin the work of rebuilding the damaged tracks and overhead wires.
"It is a significant undertaking that could take days to complete," MTA said in a statement.
The NTSB has allowed Metro-North to begin removing some of the track and wire from the scene.
Gov. Dannel P. Malloy said commuters should make plans for alternative travel through the area and urged them to consult the state Department of Transportation website for information.
Weener said data recorders on board are expected to provide the speed of the trains at the time of the crash and other information.
"Our mission is to understand not just what happened but why it happened and determine ways of preventing it from happening again," Weener said.
About 700 people were on board the Metro-North trains when one heading east from New York City's Grand Central Terminal to New Haven derailed at about 6:10 p.m. just outside Bridgeport, transit and Bridgeport officials said. Passengers described a chaotic, terrifying scene of crunching metal and flying bodies.
A spokeswoman for St. Vincent Medical Center said late Saturday that 46 people from the crash were treated there, with six of them admitted. All were in stable condition, she said.
A Bridgeport Hospital spokesman said 26 people from the crash were treated there, with three of them admitted. One was in critical condition and two were in stable condition, he said. The other 23 were released.
Bridgeport Mayor Bill Finch said the disruption caused by the crash could cost the region's economy millions of dollars.
Passenger Frank Bilotti said he was returning from a business trip in Boston on the westbound train when it crashed.
"Everybody was pretty much tossed around," said Bilotti, 53, of Westport, who wasn't injured other than a sore neck.
Firefighters used ladders to help people evacuate after the derailed cars dug into the banks of the tracks, Bilotti said.
"There were people on stretchers," he said. "There were people lying on the ground."
Blumenthal credited first responders, saying their "quick reactions and heroic efforts undoubtedly saved lives."
The MTA operates the Metro-North Railroad, the second-largest commuter railroad in the nation. The Metro-North main lines ? the Hudson, Harlem, and New Haven ? run northward from New York City's Grand Central Terminal into suburban New York and Connecticut.
At Grand Central, Frances Liu and her family were trying to get to New Haven, where Liu is graduating from Yale. A train could get them only as far north as Stamford.
"And then we'll rent a car and drive," she said.
Liu's parents had flown in from China for her graduation and were touring the country around her commencement. But the car-rental plan could face a snag ? Liu never got a driver's license, although her parents had their Chinese licenses.
"My mom can drive. So I hope it'll be OK!" Liu said as she rushed off to decide on a train.
The last significant train collision involving Metro-North occurred in 1988 when a train engineer was killed in Mount Vernon, N.Y., when one train empty of passengers rear-ended another, railroad officials said.
___
Associated Press writers Michael Melia in Hartford, Conn., Susan Haigh in Fairfield, Conn., and Verena Dobnik in New York City contributed to this report.
Source: http://news.yahoo.com/official-broken-rail-eyed-conn-train-crash-220910803.html
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BERLIN ? Conveyor belts hum quietly, towering over piles of dark red wax. A giant mauve object that looks like a deflating balloon sprawls and sags its way across three rooms. A dark pigment circle creates the illusion of a black hole opening up in the floor. And there are lots of mirrors: convex, concave, twisting and a painted "blood mirror."
British-based artist Anish Kapoor on Friday presented his world of impressions, illusions and color in Berlin, where he has created a show for the city's Martin-Gropius-Bau museum that combines works of art dating back as far as 1988 with new pieces.
Kapoor has filled a floor of the 19th-century building with some 70 works grand and small. At the center, dominating its glass-topped atrium, stands the new "Symphony for a Beloved Sun," in which four conveyor belts protrude from the floor and walls, dumping red wax on the floor in front of a huge red disk.
Kapoor, a winner of the prestigious Turner Prize and one of the creators of the twisting red Orbit Tower that overlooks London's Olympic Stadium, was wary of giving specific meaning to his work. "I've nothing to say," he said, insisting that he has "never made a work which directly points at an overt content."
"Abstract art does have this ability ... to allow content to arise rather than to say, `this is what it means,'" he told reporters ahead of the show's opening. "It's an interplay between the viewer and the material, the stuff, the object."
Color is a key interest to Kapoor in his work, particularly dark red ? a blood-like shade that the artist said has "a visceral reality."
Throughout the show, which opens to the public Saturday and runs through Nov. 24, Kapoor's conveyor belts will drop new blobs of wax and oil paint ? changing the exhibit as it goes along, museum director Gereon Sievernich said. The cannon at the center of another major work, "Shooting into the Corner," will continue to spatter one of the building's rooms with balls of red wax.
A sprawling, sagging PVC installation titled "The Death of Leviathan" picks up where the artist left off with "Leviathan," a gigantic balloon that filled Paris' Grand Palais in 2011.
Kapoor said that the melancholy, deflating piece "speaks about, inevitably, the death of the state or the decline of the state" being experienced in Europe and beyond.
___
Online: http://tinyurl.com/8zflo3w
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Source: http://www.huffingtonpost.com/2013/05/17/anish-kapoor-exhibit-arti_n_3292170.html
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Periodically, albums become available for live streaming on iTunes prior to their official release date. The hope is that not only do consumers get a chance to hear the album before buying it, but also that by offering a free and legal way to listen to the album before it?s available, there will be less motivation for eager fans to pirate leaked albums. With unreleased albums from Daft Punk and The National currently streaming on iTunes, 9to5Mac has discovered that the streams are being left completely unprotected, offering an easy way for pirates to get high-quality cuts of the albums before they?re officially released.
With traffic sniffing tools (such as Charles Proxy or HTTP Scoop) anybody can monitor their traffic while streaming the album which will show the URL of the M4P media file being streamed. Using this URL, users can easily save the album to their computer for future listening. While it?s true somebody could also just record the stream from their computer as they listen to it, it would lose some of the quality. Downloading the stream directly offers a crisp 256kbps AAC recording. It?s also true that rather than ending up with individual tracks that a listener can easily navigate through, you?re stuck with a single file that contains the whole album. You could split the album up into separate tracks yourself, but at that point it would be less effort for most people to just go pirate the album elsewhere. In fact, an illegal download of Daft Punk?s Random Access Memory currently available on a popular torrent site appears to have come from the iTunes M4P stream.
Admittedly, even if the streams were protected, piracy would still be happening. There are some people who just don?t want to pay for music. However, Apple?s handing these albums to pirates on a silver platter by offering up an unprotected, high-quality streams like this ahead of their release dates. Ironically, Apple has documentation available for developers that covers how to encrypt HTTP audio and video streams to protect from this sort of thing.
Source: 9to5Mac
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Source: http://feedproxy.google.com/~r/TheIphoneBlog/~3/KvTtJaoawLo/story01.htm
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